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Carboni Jm
Observed names:
J M Carboni,
JM Carboni
Institutions:
Mayo Clinic,
Bristol-Myers Squibb (United States)
Observed institutions:
Mayo Clinic,
Bristol-Myers Squibb (United States)
OpenAlex ID:
A5020211565
Bisubstrate inhibitors of farnesyltransferase: a novel class of specific inhibitors of ras transformed cells.
1995
·
V Manne
, N Yan
, et al.
·
PubMed
·
131
Farnesyltransferase inhibitors are inhibitors of Ras but not R-Ras2/TC21, transformation.
1995
·
J M Carboni
, N Yan
, et al.
·
PubMed
·
63
Taxol and lipopolysaccharide activation of a murine macrophage cell line and induction of similar tyrosine phosphoproteins.
1993
·
J M Carboni
, C Singh
, et al.
·
PubMed
·
26
CD14 dependent and independent signaling pathways in murine macrophages from normal and CD14 "knockout" (CD14KO) mice stimulated with LPS or taxol.
1998
·
S N Vogel
, P Y Perera
, et al.
·
PubMed
·
16
Drug efflux by breast cancer resistance protein (BCRP) is a mechanism of resistance to the insulin-like growth factor receptor/insulin receptor inhibitor, BMS-536924.
2009
·
P Haluska
, JM Carboni
, et al.
·
Cancer Research
·
3
Regulation of lymphocyte responses in vitro. VI. -Potentiation of the response to phytohemagglutinin by cytochalasin B.
1976
·
D P Bernard
, J M Carboni
, et al.
·
PubMed
·
3
P2-11-11: IGFBP Ratio Confers Resistance to IGF Targeting and Correlates with Increased Invasion and Poor Outcome in Breast Tumors.
2011
·
MA Becker
, X Hou
, et al.
·
Cancer Research
·
2
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Works count:
7
Citation count:
244
H-index:
4
I10-index:
4
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