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Feng Hao
Observed name:
Feng B. Hao
Observed institutions:
Ginkgo Bioworks, Inc. (United States),
CanSino (China)
ORCID:
0000-0002-6057-1097
Observed ORCID:
0000-0002-6057-1097
OpenAlex ID:
A5040251310
Beyond KRAS(G12C): Biochemical and Computational Characterization of Sotorasib and Adagrasib Binding Specificity and the Critical Role of H95 and Y96
2024
·
Randa Mahran
, Jonas N. Kapp
, et al.
·
ACS Chemical Biology
·
23
PDF
Design, Synthesis, and Biological Evaluation of Novel Fms-Like Tyrosine Kinase 3/VEGFR2/Histone Deacetylase Inhibitors for the Treatment of Acute Myeloid Leukemia
2025
·
Hua Tian
, Yichen Liu
, et al.
·
Journal of Medicinal Chemistry
·
17
Co-occurring EGFR p.E709X Mutation Mediates Primary Resistance to the Third-Generation EGFR-TKIs in EGFR p.G719X-Mutant Patients with Advanced NSCLC
2024
·
Lanlan Pang
, Yihua Huang
, et al.
·
Clinical Cancer Research
·
12
PDF
The Oncogenic EGFR–SHC1 Fusion Confers Insensitivity to EGFR-TKIs via Dual Activation of N-EGFR Kinase Domain and C-SHC1 Phosphorylation Sites in Lung Cancer
2026
·
Jiani Zheng
, Shen Zhao
, et al.
·
Cancer Discovery
·
2
Abstract 5679: A novel approach for antibody affinity maturation using AI-driven mutation prediction
2025
·
Yiran Li
, Hao Peng
, et al.
·
Cancer Research
·
2
Abstract 368: Neutralizing antibody against GDF-15 restores cachexia
2023
·
Feng He
, Hao Peng
, et al.
·
Cancer Research
·
2
Abstract 5981: Over 200! The largest BaF3-EGFR engineering cell lines collection, a useful platform for novel drug discovery
2025
·
Yao Tang
, Guoqian Wang
, et al.
·
Cancer Research
·
1
Abstract 4189: CRISPR-edited cell lines targeting key molecular pathways: Accelerating the development of synthetic lethal therapeutics beyond PARP inhibitors
2025
·
Yue Huang
, Yunpeng Zhai
, et al.
·
Cancer Research
·
1
Abstract 6365: The bispecific antibody KA-3008, targeting both EGFR and B7H3, increases internalization and minimizes on-target, off-tumor toxicity
2024
·
Guojin Wu
, Li Ding
, et al.
·
Cancer Research
·
1
Abstract 1677: ASPC-1-MRTX-1133R as a useful cell line model for KRAS-G12D inhibitor development
2023
·
Guoqian Wang
, Yao Tang
, et al.
·
Cancer Research
·
1
Abstract 593: Bispecific antibody against TIGIT and PVRIG enhances antitumor immunity
2023
·
Jiabei Liang
, Xiao Gao
, et al.
·
Cancer Research
·
1
Abstract 6207: Ba/F3 kinase mutant cell panel, a powerful cell model for the discovery and development of inhibitors for kinase specific mutation
2022
·
Yu Wang
, Yao Peng
, et al.
·
Cancer Research
·
1
Bispecific antibodies targeting TL1A and IL-12/23p40 suppress pathogenic Th1/Th17 responses and ameliorate colitis in mice
2026
·
Xia Li
, Hao Peng
, et al.
·
iScience
Supplementary Table S3 from The Oncogenic EGFR–SHC1 Fusion Confers Insensitivity to EGFR-TKIs via Dual Activation of N-EGFR Kinase Domain and C-SHC1 Phosphorylation Sites in Lung Cancer
2026
·
Jiani Zheng
, Shen Zhao
, et al.
Supplementary Table S2 from The Oncogenic EGFR–SHC1 Fusion Confers Insensitivity to EGFR-TKIs via Dual Activation of N-EGFR Kinase Domain and C-SHC1 Phosphorylation Sites in Lung Cancer
2026
·
Jiani Zheng
, Shen Zhao
, et al.
Supplementary Figure S4 from The Oncogenic EGFR–SHC1 Fusion Confers Insensitivity to EGFR-TKIs via Dual Activation of N-EGFR Kinase Domain and C-SHC1 Phosphorylation Sites in Lung Cancer
2026
·
Jiani Zheng
, Shen Zhao
, et al.
Data from The Oncogenic EGFR–SHC1 Fusion Confers Insensitivity to EGFR-TKIs via Dual Activation of N-EGFR Kinase Domain and C-SHC1 Phosphorylation Sites in Lung Cancer
2026
·
Jiani Zheng
, Shen Zhao
, et al.
Supplementary Figure S2 from The Oncogenic EGFR–SHC1 Fusion Confers Insensitivity to EGFR-TKIs via Dual Activation of N-EGFR Kinase Domain and C-SHC1 Phosphorylation Sites in Lung Cancer
2026
·
Jiani Zheng
, Shen Zhao
, et al.
Supplementary Figure S3 from The Oncogenic EGFR–SHC1 Fusion Confers Insensitivity to EGFR-TKIs via Dual Activation of N-EGFR Kinase Domain and C-SHC1 Phosphorylation Sites in Lung Cancer
2026
·
Jiani Zheng
, Shen Zhao
, et al.
Supplementary Table S4 from The Oncogenic EGFR–SHC1 Fusion Confers Insensitivity to EGFR-TKIs via Dual Activation of N-EGFR Kinase Domain and C-SHC1 Phosphorylation Sites in Lung Cancer
2026
·
Jiani Zheng
, Shen Zhao
, et al.
Supplementary Table S1 from The Oncogenic EGFR–SHC1 Fusion Confers Insensitivity to EGFR-TKIs via Dual Activation of N-EGFR Kinase Domain and C-SHC1 Phosphorylation Sites in Lung Cancer
2026
·
Jiani Zheng
, Shen Zhao
, et al.
Supplementary Figure S5 from The Oncogenic EGFR–SHC1 Fusion Confers Insensitivity to EGFR-TKIs via Dual Activation of N-EGFR Kinase Domain and C-SHC1 Phosphorylation Sites in Lung Cancer
2026
·
Jiani Zheng
, Shen Zhao
, et al.
Supplementary Figure S1 from The Oncogenic EGFR–SHC1 Fusion Confers Insensitivity to EGFR-TKIs via Dual Activation of N-EGFR Kinase Domain and C-SHC1 Phosphorylation Sites in Lung Cancer
2026
·
Jiani Zheng
, Shen Zhao
, et al.
Abstract 741: Illuminate your targets: Endogenous HiBiT knock-in monoclones—inventory + rapid custom.
2026
·
Xiaomeng Gou
, Yue Huang
, et al.
·
Cancer Research
Abstract 7537: From BTK in-situ mutation (C481S, A428D, etc.) to in vivo screening: A comprehensive platform to address drug resistance in BTK-targeted therapy
2026
·
Yao Tang
, Guoqian Wang
, et al.
·
Cancer Research
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Works count:
76
Citation count:
64
H-index:
3
I10-index:
3
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