Author

Sela Fadness

Institution: University of Minnesota

Observed institutions: University of Minnesota, Hamline University

OpenAlex ID: A5067374837

Chromosome missegregation during anaphase triggers p53 cell cycle arrest through histone H3.3 Ser31 phosphorylation

2016 · Edward H. Hinchcliffe, Charles A. Day, et al. · Nature Cell Biology · 93

The histone H3.3 K27M mutation suppresses Ser31phosphorylation and mitotic fidelity, which can directly drive gliomagenesis

2024 · Charles A. Day, Florina Grigore, et al. · Current Biology · 5

The histone H3.3 K27M mutation found in diffuse midline gliomas coordinately disrupts adjacent H3.3 Ser31 phosphorylation and the fidelity of chromosome segregation

2022 · Charles A. Day, Florina Grigore, et al. · bioRxiv (Cold Spring Harbor Laboratory) · 2

Dipg-05. Histone H3.3 K27m Impairs Ser31 Phosphorylation, Resulting in Chromosomal Instability, Loss of Cell Cycle Checkpoint Control, and Tumor Formation

2020 · Charles Day, Alyssa Langfald, et al. · Neuro-Oncology · 1

Pdtm-38. Histone H3.3 Ser31 Phosphorylation Regulates Chromosome Segregation and Cell Cycle Checkpoint Control and Is Impaired in Pediatric Gliomas

2019 · Charles Day, Alyssa Langfald, et al. · Neuro-Oncology

The Response of Histone H3.3 to Chromosomal Misalignment and Missegregation: a Novel Biochemical Pathway Correlated to Cell Cycle Arrest

2017 · Sela Fadness, Charles A. Day, et al. · The FASEB Journal

Works count: 6

Citation count: 101

H-index: 2

I10-index: 1