Author

Kwi-Hye Kim

Institution: Rutgers, The State University of New Jersey

Observed institution: Rutgers, The State University of New Jersey

OpenAlex ID: A5110225591

A Mouse Model of Classical Late-Infantile Neuronal Ceroid Lipofuscinosis Based on Targeted Disruption of the CLN2 Gene Results in a Loss of Tripeptidyl-Peptidase I Activity and Progressive Neurodegeneration

2004 · David E. Sleat, Jennifer A. Wiseman, et al. · Journal of Neuroscience · 149

Residual levels of tripeptidyl-peptidase I activity dramatically ameliorate disease in late-infantile neuronal ceroid lipofuscinosis

2008 · David E. Sleat, Mukarram El-Banna, et al. · Molecular Genetics and Metabolism · 52

Dipeptidyl-peptidase I does not functionally compensate for the loss of tripeptidyl-peptidase I in the neurodegenerative disease late-infantile neuronal ceroid lipofuscinosis

2008 · Kwi-Hye Kim, Christine T. Pham, et al. · Biochemical Journal · 5

Works count: 3

Citation count: 206

H-index: 3

I10-index: 2