Author

Chang-jiang Huang

Institution: University of California, Los Angeles

Observed institution: University of California, Los Angeles

OpenAlex ID: A5111463248

High Expression Rates of Human Islet Amyloid Polypeptide Induce Endoplasmic Reticulum Stress–Mediated β-Cell Apoptosis, a Characteristic of Humans With Type 2 but Not Type 1 Diabetes

2007 · Chang-jiang Huang, Chia-yu Lin, et al. · Diabetes · 417

Evidence for Proteotoxicity in β Cells in Type 2 Diabetes

2009 · Tatyana Gurlo, Sergey Ryazantsev, et al. · American Journal Of Pathology · 228

Rb and p130 control cell cycle gene silencing to maintain the postmitotic phenotype in cardiac myocytes

2011 · Patima Sdek, Peng Zhao, et al. · The Journal of Cell Biology · 176

Induction of endoplasmic reticulum stress-induced β-cell apoptosis and accumulation of polyubiquitinated proteins by human islet amyloid polypeptide

2007 · Chang-jiang Huang, Leena Haataja, et al. · American Journal of Physiology-Endocrinology and Metabolism · 138

β-Cell Dysfunctional ERAD/Ubiquitin/Proteasome System in Type 2 Diabetes Mediated by Islet Amyloid Polypeptide–Induced UCH-L1 Deficiency

2010 · Safia Costes, Chang-jiang Huang, et al. · Diabetes · 124

Many Commercially Available Antibodies for Detection of CHOP Expression as a Marker of Endoplasmic Reticulum Stress Fail Specificity Evaluation

2008 · Leena Haataja, Tatyana Gurlo, et al. · Cell Biochemistry and Biophysics · 29

Works count: 6

Citation count: 1,112

H-index: 6

I10-index: 6