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Elif Ikiz

Observed names: Elif İkiz, İkiz, Elif
Observed institution: Mersin Üniversitesi
ORCID: 0009-0008-6095-4590
Observed ORCID: 0009-0008-6095-4590
OpenAlex ID: A5117383238
RIPK1/RIPK3/MLKL Necrosome Contributes to the Sepsis-Induced Cardiorenal Necroptotic Inflammatory Injury and Mortality
2025 · Bahar Tunctan, Muhammed Ahmed-Reda Elosman, et al. · Current Molecular Pharmacology · 8
Urotensin-II receptor contributes to the pro-inflammatory TLR4/MyD88 NF-κB/iNOS/NO pathway-mediated cardiovascular response to systemic lipopolysaccharide challenge in a septic shock model in rats
2025 · Muhammed Ahmed-Reda Elosman, Sefika Pinar Senol, et al. · Cellular and Molecular Biology · 1
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Inhibition of NOX2 Prevents Impairment of Cardiovascular Response and Inflammatory Tissue Injury Mediated by Nitrosative/ Oxidative Stress in the Lipopolysaccharide-Induced Septic Shock Model in Rats
2026 · Lutfiye Kara, Muhammed Ahmed-Reda Elosman, et al. · Current Molecular Medicine
Effects of RIPK1 inhibitor necrostatin-1s on cardiovascular responses and inflammatory damage in a bilateral hind limb I/R injury model in rats
2026 · Elif Ikiz, Muhammed Ahmed-Reda Elosman, et al. · Pharmacia
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Effects of the Urotensin-ii Receptor Antagonist Sb-710411 on Remote Ischemic Preconditioning in a Rat Model of Lps-induced Endotoxic Shock: Cardiovascular Response, Inflammatory Injury, and Survival
2026 · Parinaz Amini Didani, Muhammed Ahmed-Reda Elosman, et al. · Shock
Inhibition of NOX2 prevents nitrosative/oxidative stress-mediated impaired cardiovascular response and inflammatory tissue injury in LPS-induced septic shock model in rats
2026 · Kara, Lütfiye, Ahmed-Reda, Elosman Muhammed, et al. · Mersin Üniversitesi Açık Erişim Sistemi/Mersin University Open Access System
Investigation Of The Effect Of Ripk1 İnhibition On Blood Pressure, Heart Rate, And Necroinflammatory Changes İn Cardiac, Renal, Pulmonary, Hepatic, And Cerebral Tissues İn A Bilateral Hind Limb İschemia/Reperfusion İnjury Model İn Rats
2025 · İkiz, Elif, Tunçtan, Danışman: Prof. Dr. Bahar · Mersin Üniversitesi Açık Erişim Sistemi/Mersin University Open Access System
RIPK1 inhibition prevents hypotension and tachycardia mediated by the IKKa/β/g/IkB-a/NF-kB-related pro-inflammatory pathways in a hind limb ischemia/reperfusion injury model in rats (Sıçanlarda oluşturulan arka bacak iskemi/reperfüzyon zedelenmesi modelinde RIPK1 inhibisyonu IKKa/β/g/IkB-a/NF-kB ile ilişkili pro-enflamatuvar yolların aracılık ettiği hipotansiyon ve taşikardiyi önlemektedir)
2025 · İkiz, Elif, Ahmed-Reda, Elosman Muhammed, et al. · Mersin Üniversitesi Açık Erişim Sistemi/Mersin University Open Access System
The multitarget tyrosine kinase inhibitor sunitinib prevents oxidative/nitrosative stress and acute inflammation-mediated hemodynamic disturbances in a rat model of LPS-ınduced septic shock (Çok hedefli tirozin kinaz inhibitörü sunitinib, sıçanlarda LPS ile oluşturulan septik şok modelinde oksidatif/nitrozatif stres ve akut enflamasyon aracılıklı hemodinamik bozuklukları önlemektedir)
2025 · Şenol, Şefika Pınar, Ahmed-Reda, Elosman Muhammed, et al. · Mersin Üniversitesi Açık Erişim Sistemi/Mersin University Open Access System
RIPK1/RIPK3/MLKL necrosome contributes to the nitrosative stress-mediated cardiorenal injury and mortality in lipopolysaccharide-induced sepsis model in mice (Lipopolisakkarit ile farelerde oluşturulan sepsis modelinde nitrozatif stres aracılıklı enflamatuvar kardiyorenal zedelenme ve mortaliteye RIPK1/RIPK3/MLKL nekrozomu katkıda bulunmaktadır)
2024 · Tunçtan, Bahar, Ahmed-Reda, Elosman Muhammmed, et al. · Mersin Üniversitesi Açık Erişim Sistemi/Mersin University Open Access System
Urotensin-II receptor contributes to the nitric oxide-mediated hypotension in lipopolysaccharide-induced septic shock model in rats (Lipopolisakkarit ile sıçanlarda oluşturulan septik şok modelinde nitrik oksit aracılıklı hipotansiyona ürotensin-II reseptörü katkıda bulunmaktadır)
2024 · Ahmed-Reda, Elosman Muhammmed, Şenol, Şefika Pınar, et al. · Mersin Üniversitesi Açık Erişim Sistemi/Mersin University Open Access System
Works count: 11
Citation count: 9
H-index: 1
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