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AG-58812

Funded outputs count: 10
Funder award ID: AG-58812
OpenAlex ID: G554714994
Inducible chronic NAD deficiency in mice reveals multi-systemic phenotypical, metabolic, and transcriptional changes
2026 · Andres Benitez-Rosendo, Morgan B. Feuz, et al. · Nature Communications
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Ecto-CD38-NADase inhibition modulates cardiac metabolism and protects mice against doxorubicin-induced cardiotoxicity
2024 · Thais R Peclat, Guillermo Agorrody, et al. · Cardiovascular Research · 9
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Endogenous metabolism in endothelial and immune cells generates most of the tissue vitamin B3 (nicotinamide)
2022 · Julianna D. Zeidler, Claudia C.S. Chini, et al. · iScience · 10
Mitochondrial dysfunction in cell senescence and aging
2022 · Satomi Miwa, Sonu Kashyap, et al. · Journal of Clinical Investigation · 1,064
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The CD38 glycohydrolase and the NAD sink: implications for pathological conditions
2022 · Julianna D. Zeidler, Kelly A. Hogan, et al. · American Journal of Physiology-Cell Physiology · 88
Cancer treatment-induced NAD+ depletion in premature senescence and late cardiovascular complications
2022 · Priyanka Banerjee, Elizabeth A. Olmsted-Davis, et al. · The Journal of Cardiovascular Aging · 10
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Implications of the NADase CD38 in COVID pathophysiology
2021 · Julianna D. Zeidler, Sonu Kashyap, et al. · Physiological Reviews · 28
Targeting CD38-dependent NAD+ metabolism to mitigate multiple organ fibrosis
2020 · Bo Shi, Wenxia Wang, et al. · iScience · 72
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The Multi-faceted Ecto-enzyme CD38: Roles in Immunomodulation, Cancer, Aging, and Metabolic Diseases
2019 · Kelly A. Hogan, Claudia C. S. Chini, et al. · Frontiers in Immunology · 339
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The NADase CD38 is induced by factors secreted from senescent cells providing a potential link between senescence and age-related cellular NAD+ decline
2019 · Claudia Chini, Kelly A. Hogan, et al. · Biochemical and Biophysical Research Communications · 138
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