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S2016-3
Funder:
Japan Society for the Promotion of Science
Funded outputs count:
10
Funder award ID:
S2016-3
OpenAlex ID:
G6541536851
Endothelial SIRT3 regulates myofibroblast metabolic shifts in diabetic kidneys
2021
·
Swayam Prakash Srivastava
, Jinpeng Li
, et al.
·
iScience
·
76
PDF
The PKM2 activator TEPP‐46 suppresses kidney fibrosis via inhibition of the EMT program and aberrant glycolysis associated with suppression of HIF‐1α accumulation
2020
·
Haijie Liu
, Yuta Takagaki
, et al.
·
Journal of Diabetes Investigation
·
104
PDF
Endothelial FGFR1 (Fibroblast Growth Factor Receptor 1) Deficiency Contributes Differential Fibrogenic Effects in Kidney and Heart of Diabetic Mice
2020
·
Jinpeng Li
, Haijie Liu
, et al.
·
Hypertension
·
74
PDF
CD‐1db/db mice: A novel type 2 diabetic mouse model with progressive kidney fibrosis
2020
·
Yuiko Mizunuma
, Keizo Kanasaki
, et al.
·
Journal of Diabetes Investigation
·
14
PDF
Dipeptidyl peptidase-4 plays a pathogenic role in BSA-induced kidney injury in diabetic mice
2019
·
Yuta Takagaki
, Sen Shi
, et al.
·
Scientific Reports
·
28
PDF
βklotho is essential for the anti‐endothelial mesenchymal transition effects of N‐acetyl‐seryl‐aspartyl‐lysyl‐proline
2019
·
Rongfen Gao
, Keizo Kanasaki
, et al.
·
FEBS Open Bio
·
14
PDF
SIRT3 deficiency leads to induction of abnormal glycolysis in diabetic kidney with fibrosis
2018
·
Swayam Prakash Srivastava
, Jinpeng Li
, et al.
·
Cell Death and Disease
·
174
PDF
Ipragliflozin improves mitochondrial abnormalities in renal tubules induced by a high‐fat diet
2018
·
Susumu Takagi
, Jinpeng Li
, et al.
·
Journal of Diabetes Investigation
·
127
PDF
FGFR1 is essential for N-acetyl-seryl-aspartyl-lysyl-proline regulation of mitochondrial dynamics by upregulating microRNA let-7b-5p
2017
·
Qiongying Hu
, Jinpeng Li
, et al.
·
Biochemical and Biophysical Research Communications
·
15
FGFR1 is critical for the anti-endothelial mesenchymal transition effect of N-acetyl-seryl-aspartyl-lysyl-proline via induction of the MAP4K4 pathway
2017
·
Jinpeng Li
, Sen Shi
, et al.
·
Cell Death and Disease
·
68
PDF
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