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HIF1α Represses Cell Stress Pathways to Allow Proliferation of Hypoxic Fetal Cardiomyocytes

Year: 2015
Type: article
DOI: 10.1016/j.devcel.2015.04.021
Authors: Nuno Guimarães-Camboa, Jennifer Stowe, Ivy Aneas, Noboru Sakabe, Paola Cattaneo, Lindsay Henderson, Michael S. Kilberg, Randall S. Johnson, Ju Chen, Andrew D. McCulloch, Marcelo A. Nobrega, Sylvia M. Evans, Alexander C. Zambon
Institutions: Universidade do Porto, University of California San Diego, Instituto de Ciências Biomédicas Albel Salazar, University of Chicago, University of Florida Health, University of Florida, UF Health Cancer Center, University of Cambridge, Keck Graduate Institute
Language: English
Open Access status: bronze
Funders: American Heart Association, Wellcome Trust, National Institutes of Health, University of California, San Diego, National Heart, Lung, and Blood Institute, National Institute of General Medical Sciences, National Institute of Diabetes and Digestive and Kidney Diseases, National Institute of Neurological Disorders and Stroke
Awards: G1331601629, Molecular mechanisms of hypoxia tolerance and susceptibility, G2112935040, G3033040839, G3367681203, G3713841663, G3939850859, G4209006228, G4460446700, The physiology of hypoxic response, G5451227829, G5863040294, University of California San Diego Neuroscience Microscopy Imaging Core, G6364856392, G639426234, Nesprin 1 and 2 in cardiac structure, function, and disease, The Role of Cypher and ENH in heart, G752722152, Approaches to Cardiac Regeneration, Amino Acid Regulation of Alternative Splicing, Approaches to Cardiac Regeneration, G8971701765, G9000075628, G9000151822, G9000160049, G9000255603, San Diego IRACDA Scholars Program
OpenAlex ID: W1523978214
FWCI: 3.986
Cites: 30
Cited by: 153
Related to: 10