A functionally inactive p53 protein interatocarcinoma cells is activated by either DNA damage or cellular differentiation
Year: 1996
Type: article
Source: Nature Medicine
DOI: 10.1038/nm0796-804
Authors: Stuart G. Lutzker, Arnold J. Levine
Institutions: Princeton University, Rutgers Cancer Institute
Language: English
Subfield: Oncology
Field: Medicine
Domain: Health Sciences
Open Access status: closed
Funder: U.S. Public Health Service
Award: G5457356855
OpenAlex ID: W1971215274
