Work

The t(8;21) fusion protein, AML1–ETO, specifically represses the transcription of the p14ARF tumor suppressor in acute myeloid leukemia

Year: 2002

Type: article

Source: Nature Medicine

DOI: 10.1038/nm726

Authors: Bryan Linggi, Carsten Müller-Tidow, Louis van de Locht, Ming Hu, John Nip, Hubert Serve, Wolfgang E. Berdel, Bert van der Reijden, Dawn E. Quelle, Janet D. Rowley, John Cleveland, Joop H. Jansen, Pier Paolo Pandolfi, Scott W. Hiebert

Institutions: Vanderbilt University, Vanderbilt University Medical Center, University of Münster, University Hospital Münster, Radboud University Nijmegen, Radboud University Medical Center, Memorial Sloan Kettering Cancer Center, Cornell University, University of Iowa, University of Chicago, St. Jude Children's Research Hospital

Language: English

Topic: Acute Myeloid Leukemia Research

Subfield: Hematology

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: closed

Funders: National Institutes of Health, U.S. Public Health Service, National Institute on Aging, National Cancer Institute

Awards: E2F AND DIFFERENTIATION, APOPTOSIS, AND LEUKEMIA, G4513176822, G5267996292, G6124872711, Role of AML1-ETO in Acute Leukemia, Cancer Center Support Grant, G963484308

OpenAlex ID: W1973023394

FWCI: 10.4

Cites: 44

Cited by: 259

Related to: 10