Work

Loss of OPA1 disturbs cellular calcium homeostasis and sensitizes for excitotoxicity

Year: 2012

Type: article

Source: Cell Death and Differentiation

DOI: 10.1038/cdd.2012.128

Authors: Y E Kushnareva, A A Gerencser, B Bossy, W-K Ju, A D White, J Waggoner, M H Ellisman, G Perkins, E Bossy-Wetzel

Institutions: Sanford Burnham Prebys Medical Discovery Institute, Buck Institute for Research on Aging, University of Central Florida, University of California San Diego

Language: English

Topic: Mitochondrial Function and Pathology

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: bronze

Funders: National Institutes of Health, National Eye Institute, National Institute of Neurological Disorders and Stroke, National Center for Research Resources

Awards: IVEM and Image Analysis Resource, DYNAMICS OF MEMBRANE ORGANIZATION AT NODE OF RANVIER, Mitochondrial Fission and Neuronal Cell Death, Mitochondrial fission in Huntington's Disease, Mitochondrial Dysfunction in Glaucomatous Optic Neuropathy, OPA1 Mutation and Retinal Ganglion Cell Degeneration

OpenAlex ID: W1975562298

FWCI: 3.083

Cites: 59

Cited by: 115

Related to: 10