Work

ADAM10 Missense Mutations Potentiate β-Amyloid Accumulation by Impairing Prodomain Chaperone Function

Year: 2013

Type: article

Source: Neuron

DOI: 10.1016/j.neuron.2013.08.035

Authors: Jaehong Suh, Se Hoon Choi, Donna M. Romano, Moira A. Gannon, Andrea N. Lesinski, Doo Yeon Kim, Rudolph E. Tanzi

Institutions: Harvard University, Massachusetts General Hospital

Language: English

Topic: Alzheimer's disease research and treatments

Subfield: Physiology

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: bronze

Funders: National Institute on Aging, National Institute of Mental Health, National Institute of Neurological Disorders and Stroke

Awards: Identification of Functional Genomic Variants in Alzheimer's Disease, Characterization of Alzheimer's Mutations in ADAM10., Massachusetts Alzheimer's Disease Research Center, Role of MAMs in stabilization and BACE1-mediated processing of palAPP., Massachusetts Alzheimer's Disease Research Center (MADRC)

OpenAlex ID: W1979909831

FWCI: 14.6

Cites: 49

Cited by: 217

Related to: 10