Work

The API2–MALT1 fusion exploits TNFR pathway-associated RIP1 ubiquitination to promote oncogenic NF-κB signaling

Year: 2013

Type: article

Source: Oncogene

DOI: 10.1038/onc.2013.195

Authors: S Rosebeck, A O Rehman, I J Apel, D Kohrt, A Appert, M A O'Donnell, A T Ting, M-Q Du, M Baens, P C Lucas, L M McAllister-Lucas

Institutions: University of Michigan, Michigan Medicine, University of Cambridge, Addenbrooke's Hospital, Icahn School of Medicine at Mount Sinai, Vlaams Instituut voor Biotechnologie, KU Leuven

Language: English

Topic: NF-κB Signaling Pathways

Subfield: Cancer Research

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: green

Funders: University of Michigan, University of Michigan Comprehensive Cancer Center, National Heart, Lung, and Blood Institute, National Cancer Institute

Awards: GPCR Signaling Through a Novel NF-kB Pathway, Multiple Roles of the API2 Moiety in API2-MALT1-Mediated Lymphomagenesis, Boxer Training Program in Hematology

OpenAlex ID: W1996738870

FWCI: 0.907

Cites: 54

Cited by: 29

Related to: 10