Work

Acetylation of the c-MYC oncoprotein is required for cooperation with the HTLV-1 p30II accessory protein and the induction of oncogenic cellular transformation by p30II/c-MYC

Year: 2015

Type: article

Source: Virology

DOI: 10.1016/j.virol.2014.12.008

Authors: Megan M. Romeo, Bookyung Ko, Janice Kim, Rebecca Brady, Hayley C. Heatley, Jeffrey He, Carolyn K. Harrod, Braden Barnett, Lee Ratner, Michael D. Lairmore, Ernest Martinez, Bernhard Lüscher, Craig N. Robson, Marie Henriksson, Robert Harrod

Institutions: Southern Methodist University, Washington University in St. Louis, University of California, Davis, University of California, Riverside, Universitätsklinikum Aachen, RWTH Aachen University, Newcastle University, Karolinska Institutet

Language: English

Topic: T-cell and Retrovirus Studies

Subfield: Immunology

Field: Immunology and Microbiology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: bronze

Funders: University of Washington, National Institutes of Health, National Cancer Institute

Awards: PROLIFERATIVE EFFECTS OF HTLV-1, G4533449836, Modulation of c-MYC Functions in an HTLV-1 Model for Carcinogenesis, G5945563202, G790105675, G8767817759, Retrovirus Models of Cancer, G964580632

OpenAlex ID: W2000539064

FWCI: 0.687

Cites: 106

Cited by: 18

Related to: 10