Work

Telomere shortening and loss of self-renewal in dyskeratosis congenita induced pluripotent stem cells

Year: 2011

Type: article

Source: Nature

DOI: 10.1038/nature10084

Authors: Luis F. Z. Batista, Matthew F. Pech, Franklin L. Zhong, Ha Nam Nguyen, Kathleen T. Xie, Arthur J. Zaug, Sharon M. Crary, Jinkuk Choi, Vittorio Sebastiano, Athena Cherry, Neelam Giri, Marius Wernig, Blanche P. Alter, Thomas R. Cech, Sharon A. Savage, Renee A. Reijo Pera, Steven E. Artandi

Institutions: Stanford Medicine, Stanford University, Institute for Stem Cell Biology and Regenerative Medicine, Howard Hughes Medical Institute, University of Colorado Boulder, National Institutes of Health, National Cancer Institute, Division of Cancer Epidemiology and Genetics

Language: English

Topic: Telomeres, Telomerase, and Senescence

Subfield: Physiology

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: green

Funders: National Science Foundation, Howard Hughes Medical Institute, National Institutes of Health, National Institute on Aging, National Heart, Lung, and Blood Institute, National Cancer Institute

Awards: Regulation of the Telomerase Protein Component In Cancer, Basic and Translational Research iPSC-based hematologic and vascular therapies, Defining the telomerase holoenzyme in progenitor cells with aging, Studying a Human Stem Cell Disease Using iPS Technology, Telomeres, Telomerase and Tumor Progression, Leveraging innovative technologies in basic and clinical cancer research

OpenAlex ID: W2012918968

FWCI: 21.54

Cites: 37

Cited by: 260

Related to: 10