Work

Disease‐associated casein kinase I δ mutation may promote adenomatous polyps formation via a Wnt/β‐catenin independent mechanism

Year: 2006

Type: article

Source: International Journal of Cancer

DOI: 10.1002/ijc.22368

Authors: I‐Chun Tsai, Margaret Woolf, Deborah W. Neklason, William W. Branford, H. Joseph Yost, Randall W. Burt, David M. Virshup

Institutions: University of Utah, Huntsman Cancer Institute

Language: English

Topic: Wnt/β-catenin signaling in development and cancer

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: closed

Funders: National Institutes of Health, National Cancer Institute

Awards: G1192507251, G210865433, HIGH RISK FAMILIAL COLON CANCER- GENETICS AND PHENOTYPE, G4967445609, G5129853025, Molecular and Clinical Approaches to Colon Cancer Precursors, G623163788, REGULATIONS OF BETA CATENIN BY PROTEIN PHOSPHATASE 2A, Cancer Center Support Grant

OpenAlex ID: W2014626328

FWCI: 0.491

Cites: 50

Cited by: 30

Related to: 10