Work

ATG12–ATG3 interacts with Alix to promote basal autophagic flux and late endosome function

Year: 2015

Type: article

Source: Nature Cell Biology

DOI: 10.1038/ncb3112

Authors: Lyndsay Murrow, Ritu Malhotra, Jayanta Debnath

Institutions: University of California, San Francisco, UCSF Helen Diller Family Comprehensive Cancer Center

Language: English

Topic: Autophagy in Disease and Therapy

Subfield: Epidemiology

Field: Medicine

Domain: Health Sciences

Open Access status: bronze

Funders: National Science Foundation, Howard Hughes Medical Institute, National Institutes of Health, University of California, San Francisco, National Cancer Institute

Awards: G4288435686, Autophagy as a microenvironmental regulator of tumorigenesis and resistance, Graduate Research Fellowship Program (GRFP), G8722063621, Autophagy and Pro-metastatic Differentiation

OpenAlex ID: W2015781356

FWCI: 16.31

Cites: 55

Cited by: 301

Related to: 10