Work

Hepatic Insulin Resistance Is Sufficient to Produce Dyslipidemia and Susceptibility to Atherosclerosis

Year: 2008

Type: article

Source: Cell Metabolism

DOI: 10.1016/j.cmet.2007.11.013

Authors: Sudha B. Biddinger, Antonio Hernandez-Ono, Christian Rask-Madsen, Joel T. Haas, José O. Alemán, Ryo Suzuki, Erez F. Scapa, Chhavi Agarwal, Martin C. Carey, Gregory Stephanopoulos, David E. Cohen, George L. King, Henry N. Ginsberg, C. Ronald Kahn

Institutions: Joslin Diabetes Center, Harvard University, Columbia University, Massachusetts Institute of Technology, Brigham and Women's Hospital

Language: English

Topic: Lipid metabolism and disorders

Subfield: Cardiology and Cardiovascular Medicine

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: bronze

Funders: National Heart, Lung, and Blood Institute, National Institute of Diabetes and Digestive and Kidney Diseases

Awards: Insulin Receptor Structure and Turnover, G1779690066, The Interaction Between Hepatic Insulin Resistance and Cholesterol Metabolism, Fatty Acid Regulation of Liver Lipoprotein Production, Insulin Receptor Structure and Turnover, G2248290576, Selective Insulin Resistance- Diabetic Cardiovascular Diseases, G2631439768, G2758369363, G2810564815, G390542385, Vanderbilt Mouse Metabolic Phenotyping Center, G4138677399, G508144266, Molecular Pathogenesis of Cystic Fibrosis Liver Disease, GENE EXPRESSION IN DIABETES BY SUBTRACTIVE CLONING, Phospholipid-Mediated Metabolic Control in the Pathogenesis of NAFLD, Career Development in Diabetes for Pediatric Endocrinolo, Regulation of Hepatic Lipid and Glucose Metabolism by PC-TP/StarD2, Elucidating modulators of hepatic metabolism by quantitative flux analysis, G8179588267, Diabetes Research Center, Lipid-Mediated Control of Acetyl-CoA Metabolism in the Pathogenesis of NAFLD

OpenAlex ID: W2019973807

FWCI: 30.32

Cites: 80

Cited by: 467

Related to: 10