Work

Targeted Deletion of Thioredoxin-Interacting Protein Regulates Cardiac Dysfunction in Response to Pressure Overload

Year: 2007

Type: article

Source: Circulation Research

DOI: 10.1161/circresaha.106.160515

Authors: Jun Yoshioka, Kenichi Imahashi, Scott A. Gabel, William A. Chutkow, Aurora A. Burds, Joseph Gannon, P. Christian Schulze, Catherine MacGillivray, Robert E. London, Elizabeth Murphy, Richard T. Lee

Institutions: Brigham and Women's Hospital, Harvard University, National Institute of Environmental Health Sciences, Center for Cancer Research, Massachusetts Institute of Technology

Language: English

Topic: Cardiac Fibrosis and Remodeling

Subfield: Cardiology and Cardiovascular Medicine

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: closed

Funders: National Heart, Lung, and Blood Institute, National Institute of General Medical Sciences

Awards: G2835674304, G3577149824, VDUP1/thioredoxin and cardiomyocyte stress, Arterial Dysfunction: Basic and Clinical Mechanisms, G868400512

OpenAlex ID: W2021031416

FWCI: 6.32

Cites: 28

Cited by: 115

Related to: 10