Work

Overexpression of Transforming Growth Factor β1 in Malignant Prostate Cells is Partly Caused by a Runaway of TGF-β1 Auto-induction Mediated Through a Defective Recruitment of Protein Phosphatase 2A by TGF-β Type I Receptor

Year: 2010

Type: article

Source: Urology

DOI: 10.1016/j.urology.2010.03.061

Authors: Nengwang Yu, James M. Kozlowski, Irwin I. Park, Lin Chen, Qiang Zhang, Danfeng Xu, Jennifer A. Doll, Susan E. Crawford, Charles B. Brendler, Chung Lee

Institutions: Northwestern University, Second Military Medical University, Shanghai Changzheng Hospital, Robert H. Lurie Comprehensive Cancer Center of Northwestern University, Endeavor Health

Language: English

Topic: TGF-β signaling in diseases

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: green

Funders: U.S. Department of Defense, NorthShore University HealthSystem, China Scholarship Council, National Institutes of Health, National Cancer Institute

Awards: G2927653515, SPORE in Prostate Cancer

OpenAlex ID: W2022094271

FWCI: 1.035

Cites: 34

Cited by: 31

Related to: 10