Work

Progressive phenotype and nuclear accumulation of an amino-terminal cleavage fragment in a transgenic mouse model with inducible expression of full-length mutant huntingtin

Year: 2005

Type: article

Source: Neurobiology of Disease

DOI: 10.1016/j.nbd.2005.07.014

Authors: Yuji Tanaka, Shuichi Igarashi, Masayuki Nakamura, Juliette Gafni, Cameron Torcassi, Gabrielle Schilling, Danielle Crippen, Jonathan D. Wood, Akira Sawa, Nancy A. Jenkins, Neal G. Copeland, David R. Borchelt, Christopher A. Ross, Lisa M. Ellerby

Institutions: Johns Hopkins University, Johns Hopkins Medicine, Buck Institute for Research on Aging, National Cancer Institute

Language: English

Topic: Genetic Neurodegenerative Diseases

Subfield: Cellular and Molecular Neuroscience

Field: Neuroscience

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: green

Funders: Hereditary Disease Foundation, Cure Huntington's Disease Initiative, National Institutes of Health, National Institute of Neurological Disorders and Stroke

Awards: G10698365, G2137558941, Triplet Repeat Disease:Requirement for Caspase Cleavage, G288079080, Calpain in Huntington's Disease, G3645877733, Neurodegeneration and Polyglutamine Toxicity DRPLA, Transgenic Models of Huntington's Disease

OpenAlex ID: W2027280805

FWCI: 1.685

Cites: 64

Cited by: 63

Related to: 10