Work

N17 Modifies Mutant Huntingtin Nuclear Pathogenesis and Severity of Disease in HD BAC Transgenic Mice

Year: 2015

Type: article

Source: Neuron

DOI: 10.1016/j.neuron.2015.01.008

Authors: Xiaofeng Gu, Jeffrey P. Cantle, Erin R. Greiner, C.Y. Daniel Lee, Albert M. Barth, Fuying Gao, Chang Sin Park, Zhiqiang Zhang, Susana Sandoval-Miller, Richard L. Zhang, Marc Diamond, Istvan Mody, Giovanni Coppola, X. William Yang

Institutions: University of California, Los Angeles, Washington University in St. Louis, The University of Texas Southwestern Medical Center

Language: English

Topic: Genetic Neurodegenerative Diseases

Subfield: Cellular and Molecular Neuroscience

Field: Neuroscience

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: bronze

Funder: National Institute of Neurological Disorders and Stroke

Awards: Complement-Microglia Interaction in Synaptic Loss and Neurodegeneration in HD, Conditional Models: non-cell-autonomous Toxicities in HD, Validating NT17 Dependent Mechanisms in Huntington's Disease Suppression, Integrative Center for Neurogenetics and Neurogenomics - Overall, Training Grant in Neurobehavioral Genetics

OpenAlex ID: W2029399677

FWCI: 4.124

Cites: 64

Cited by: 78

Related to: 10