Doubly mutant mice, deficient in connexin32 and -43, show normal prenatal development of organs where the two gap junction proteins are expressed in the same cells
Year: 1999
Type: article
Source: Developmental Genetics
Institutions: Western University, University of Bonn, Essen University Hospital
Language: English
Topic: Connexins and lens biology
Subfield: Molecular Biology
Domain: Life Sciences
Open Access status: closed
OpenAlex ID: W2033686650
