Work

βA3/A1-crystallin: More than a lens protein

Year: 2014

Type: article

Source: Progress in Retinal and Eye Research

DOI: 10.1016/j.preteyeres.2014.11.002

Authors: J. Samuel Zigler, Debasish Sinha

Institutions: Johns Hopkins University, Johns Hopkins Medicine, Johns Hopkins Hospital

Language: English

Topic: Connexins and lens biology

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

Open Access status: green

Funders: Knights Templar Eye Foundation, National Institutes of Health, National Eye Institute

Awards: A novel bA3/A1-crystallin gene mutation results in persistent fetal vasculature, G1582333981, A CRYSTALLIN MUTATION WITH ABNORMAL ASTROCYTES AND RETINAL VESSELS, G2795717175, G3033057233, G4716643746, P30 Wilmer Core Grant for Vision Research, G6517750212, G9000169224

OpenAlex ID: W2038569321

FWCI: 1.217

Cites: 116

Cited by: 44

Related to: 10