Work

Loss of Integrin α9β1 Results in Defects in Proliferation, Causing Poor Re-Epithelialization during Cutaneous Wound Healing

Year: 2008

Type: article

Source: Journal of Investigative Dermatology

DOI: 10.1038/jid.2008.201

Authors: Purva Singh, Chun Chen, Sonali Pal-Ghosh, Mary Ann Stepp, Dean Sheppard, Livingston Van De Water

Institutions: Albany Medical College, University of California, San Francisco, George Washington University

Language: English

Topic: Cell Adhesion Molecules Research

Subfield: Immunology and Allergy

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: bronze

Funders: Gladstone Institutes, National Institutes of Health, U.S. Public Health Service, National Heart, Lung, and Blood Institute, National Institute of General Medical Sciences

Awards: Matrix Regulation of Cell Function During Wound Healing, G2128184899, In Vivo Function of Pulmonary Integrins, G358049942, G554800201, The NHLBI-Bay Area Functional Genomics Consortium, Integrin-Mediated Development of the Thoracic Duct, G8471047364, In Vivo Function of Pulmonary Integrins, G945742809, G988875928

OpenAlex ID: W2041795914

FWCI: 4.314

Cites: 54

Cited by: 86

Related to: 10