Work

Adenosine is required for sustained inflammasome activation via the A2A receptor and the HIF-1α pathway

Year: 2013

Type: article

Source: Nature Communications

DOI: 10.1038/ncomms3909

Authors: Xinshou Ouyang, Ayaz Ghani, Ahsan Malik, Tuere Wilder, Oscar Rene Colegio, Richard Anthony Flavell, Bruce Neil Cronstein, Wajahat Zafar Mehal

Institutions: Yale University, VA Connecticut Healthcare System, New York University

Language: English

Topic: Inflammasome and immune disorders

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: bronze

Funders: Howard Hughes Medical Institute, Yale University, University of Louisville, National Institutes of Health, National Institute of Diabetes and Digestive and Kidney Diseases, National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Center for Research Resources, Yale Liver Center

Awards: G1200968478, G122403880, G1816712483, G210642380, G2555113698, G3396575264, Mechanisms of Bile Secretion and Cholestasis, G4184292084, G53965780, G6268016241, G6509518109, The Pharmacology of Dermal Fibrosis, G9000120564, G9000136759, G9000136791, G9000188258, G9000195372, G9000331982, G9000334970

OpenAlex ID: W2052663559

FWCI: 3.227

Cites: 33

Cited by: 129

Related to: 10