Work

Protease-Activated Receptor 2, Dipeptidyl Peptidase I, and Proteases Mediate Clostridium difficile Toxin A Enteritis

Year: 2007

Type: article

Source: Gastroenterology

DOI: 10.1053/j.gastro.2007.03.101

Authors: Graeme S. Cottrell, Silvia Amadesi, Stella Pikios, Eric Camerer, J. Adam Willardsen, Brett R. Murphy, George H. Caughey, Paul J. Wolters, Shaun R. Coughlin, Anders Peterson, Wolfgang Knecht, Charalabos Pothoulakis, Nigel W. Bunnett, Eileen F. Grady

Institutions: University of California, San Francisco, AstraZeneca (Sweden), Beth Israel Deaconess Medical Center, Harvard University

Language: English

Topic: Clostridium difficile and Clostridium perfringens research

Subfield: Infectious Diseases

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: green

Funders: Crohn's and Colitis Foundation of America, National Heart, Lung, and Blood Institute, National Institute of Diabetes and Digestive and Kidney Diseases

Awards: G2119311264, G2243887966, G2570620649, G3025583357, G3999202268, Evolving Microenvironments in Airway Inflammation, G4373818538, REGULATION OF CELLULAR RESPONSES TO NEUROPEPTIDES, G4788452553, Proteinase-Activated Receptors/Peripheral Nervous System, NEUROGENIC MECHANISMS OF INTESTINAL INFLAMMATION, Regulation of Signaling by Cell Surface Proteolysis, CURE: Digestive Diseases Research Center, G7463305858, G750998215, Role of Substance P in Intestinal Inflammation, REGULATION OF CELLULAR RESPONSES TO NEUROPEPTIDES, REGULATION OF SIGNALING BY CELL-SURFACE PROTEOLYSIS

OpenAlex ID: W2058871622

FWCI: 3.818

Cites: 57

Cited by: 55

Related to: 10