Work

Soluble pathological tau in the entorhinal cortex leads to presynaptic deficits in an early Alzheimer’s disease model

Year: 2013

Type: article

Source: Acta Neuropathologica

DOI: 10.1007/s00401-013-1215-5

Authors: Manuela Polydoro, Volodymyr I. Dzhala, Amy M. Pooler, Samantha B. Nicholls, A. Patrick McKinney, Laura Sanchez, Rose Pitstick, George A. Carlson, Kevin J. Staley, Tara L. Spires-Jones, Bradley T. Hyman

Institutions: Harvard University, Massachusetts General Hospital, King's College London, Harvard NeuroDiscovery Center, McLaughlin Research Institute

Language: English

Topic: Alzheimer's disease research and treatments

Subfield: Cellular and Molecular Neuroscience

Field: Neuroscience

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: green

Funders: National Institutes of Health, National Institute on Aging

Awards: Anatomical Changes in Tau Transgenic Models, Synaptic loss and plasticity in aging and Alzheimer's disease, A Model of Early Alzheimer Disease, Massachusetts Alzheimer's Disease Research Center, G7907190449, Training in the Molecular Biology of Neurodegeneration and Alzheimer's Disease, Massachusetts Alzheimer's Disease Research Center (MADRC)

OpenAlex ID: W2059127030

FWCI: 3.248

Cites: 63

Cited by: 122

Related to: 10