Work

Loss of Wdfy3 in mice alters cerebral cortical neurogenesis reflecting aspects of the autism pathology

Year: 2014

Type: article

Source: Nature Communications

DOI: 10.1038/ncomms5692

Authors: Lori A. Orosco, Adam P. Ross, Staci L. Cates, Sean E. Scott, Dennis Wu, Jiho Sohn, David Pleasure, Samuel J. Pleasure, Iannis E. Adamopoulos, Konstantinos S Zarbalis

Institutions: Shriners Hospitals for Children - Northern California, University of California, Davis, University of California, San Francisco

Language: English

Topic: Autism Spectrum Disorder Research

Subfield: Cognitive Neuroscience

Field: Neuroscience

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: bronze

Funders: Simons Foundation, Nancy Lurie Marks Family Foundation, Regeneron Pharmaceuticals, Shriners Hospitals for Children, Simons Foundation Autism Research Initiative, National Institutes of Health, U.S. Public Health Service, University of California, Davis, National Institute of Mental Health, National Human Genome Research Institute, National Center for Research Resources, Eunice Kennedy Shriver National Institute of Child Health and Human Development

Awards: G1320710445, G2024429596, Production of Targeted Null Mutations for 10,000 Genes in C57BL/6 ES Cells, Wdfy3, a novel regulator of neuronal migration and connectivity, G5816300381, Defective Forebrain Development in Mutant Mice, UCD KOMP Project Repository, High-throughput targeted mutagenesis of mouse stem cell lines

OpenAlex ID: W2059543801

FWCI: 5.021

Cites: 72

Cited by: 90

Related to: 10