Work

TP53 mutations in myeloid malignancies are either homozygous or hemizygous due to copy number-neutral loss of heterozygosity or deletion of 17p

Year: 2009

Type: article

Source: Leukemia

DOI: 10.1038/leu.2009.189

Authors: M Jasek, L P Gondek, N Bejanyan, R Tiu, J Huh, K S Theil, C O'Keefe, M A McDevitt, J P Maciejewski

Institutions: Cleveland Clinic, Ewha Womans University, Ewha Womans University Seoul Hospital, Johns Hopkins University, Johns Hopkins Medicine

Language: English

Topic: Acute Myeloid Leukemia Research

Subfield: Hematology

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: green

Funders: Helsingin ja Uudenmaan Sairaanhoitopiiri, National Institutes of Health, National Heart, Lung, and Blood Institute, National Center for Research Resources

Awards: G2564966186, G481402874, Bone Marrow Failure Clinical Research Center, Training/Research Center-Bone Marrow Failure Syndromes, Predisposition to chromosomal alterations in MDS

OpenAlex ID: W2079349819

FWCI: 4.648

Cites: 8

Cited by: 83

Related to: 10