Work

Microglial Beclin 1 Regulates Retromer Trafficking and Phagocytosis and Is Impaired in Alzheimer’s Disease

Year: 2013

Type: article

Source: Neuron

DOI: 10.1016/j.neuron.2013.06.046

Authors: Kurt M. Lucin, Caitlin E. O’Brien, Gregor Bieri, Eva Czirr, Kira I. Mosher, Rachelle J. Abbey, Diego F. Mastroeni, Joseph Rogers, Brian Spencer, Eliezer Masliah, Tony Wyss-Coray

Institutions: VA Palo Alto Health Care System, Stanford Medicine, Stanford University, Banner Sun Health Research Institute, SRI International, University of California San Diego

Language: English

Topic: Neuroinflammation and Neurodegeneration Mechanisms

Subfield: Neurology

Field: Neuroscience

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: bronze

Funders: U.S. Department of Veterans Affairs, University of California, Irvine, National Institute on Aging, National Institute of Mental Health, National Institute of Neurological Disorders and Stroke

Awards: Regulation of microglial functions by neural progenitor cell-derived factors, G164548318, a-synuclein clearance strategies for the treatment of Lewy body disease, a-Synuclein vulnerability mechanisms and therapeutics in Alzheimer's Disease, UCSD Alzheimer's Disease Research Center P50, G3701860004, Controlled Activation of Complement to Treat Alzheimer's, Stanford Neuroscience Research Cores for Gene Vectors, Microscopy, and Behavios, Beclin 1 Neurodegeneration and Alzheimer's Disease, Circulatory Rejuvenating Factors for the Brain, Arizona Alzheimer's Disease Core Center, Beclin 1 regulates neuroprotective TGF-beta signaling, National Brain and Tissue Resource for Parkinson's Disease and Related Disorders, Clearance Pathways in the CNS in Aging and HIV, Neuroscience Research Training

OpenAlex ID: W2081014274

FWCI: 13.96

Cites: 62

Cited by: 373

Related to: 10