Work

Amyloid-β protein impairs Ca2+ release and contractility in skeletal muscle

Year: 2008

Type: article

Source: Neurobiology of Aging

DOI: 10.1016/j.neurobiolaging.2008.11.003

Authors: Alexander Shtifman, Christopher W. Ward, Derek R. Laver, Mark L. Bannister, Jose R. Lopez, Masashi Kitazawa, Frank M. LaFerla, Noriaki Ikemoto, Henry W. Querfurth

Institutions: Tufts University, St. Elizabeth's Medical Center, University of Maryland, Baltimore, University of Newcastle Australia, Boston Biomedical Research Institute, Brigham and Women's Hospital, Harvard University, Instituto Venezolano de Investigaciones Científicas, University of California, Irvine

Language: English

Topic: Muscle Physiology and Disorders

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: green

Funders: National Institutes of Health, National Institute of Arthritis and Musculoskeletal and Skin Diseases

Awards: Role of RyRs, DHPRs and Ca2+ Entry in the Inclusion Body Myositis, Role of RyRs and DHPRs and Ca2+ Entry in beta-amyloid mediated Ca2+ dysregulation

OpenAlex ID: W2086121753

FWCI: 1.644

Cites: 51

Cited by: 58

Related to: 10