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Reduction of myeloid suppressor cell derived nitric oxide provides a mechanistic basis of lead enhancement of alloreactive CD4+ T cell proliferation

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Year: 2007
Type: article
Source: Toxicology and Applied Pharmacology
DOI: 10.1016/j.taap.2007.12.011
Authors: David G. Farrer, Sara Hueber, Michael D. Laiosa, Kevin G. Eckles, Michael J. McCabe Jr.
Institutions: University of Rochester Medicine, University of Rochester
Language: English

Topic: Immunotoxicology and immune responses
Subfield: Immunology
Field: Immunology and Microbiology
Domain: Life Sciences
SDG: Good health and well-being

Open Access status: green

Funders: National Institutes of Health, Medical Center, University of Rochester, National Institute of Environmental Health Sciences
Awards: G1081269979, G1862412751, G469873995, Death Receptor Signaling and Mercury Immunotoxicity, Environmental Agents as Modulators of Disease Processes
OpenAlex ID: W2086308129
FWCI: 0.219
Cites: 37
Cited by: 9
Related to: 10
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