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The C5a complement activation peptide increases IL-1β and IL-6 release from amyloid-β primed human monocytes: implications for Alzheimer’s disease

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Year: 2000
Type: article
Source: Journal of Neuroimmunology
DOI: 10.1016/s0165-5728(00)00291-5
Authors: Stephen O’Barr, Neil R Cooper
Institutions: Western University of Health Sciences, Scripps Research Institute
Language: English

Topic: Neuroinflammation and Neurodegeneration Mechanisms
Subfield: Neurology
Field: Neuroscience
Domain: Life Sciences
SDG: Good health and well-being

Open Access status: closed

Funders: National Institutes of Health, National Institute on Aging, National Heart, Lung, and Blood Institute, National Institute of Neurological Disorders and Stroke
Awards: Aging Disease--Prion/Transgenic/Immunologic Studies, G3734952186, G415628284, ROLE OF COMPLEMENT ACTIVATION IN ALZHEIMER'S DISEASE, Blood System in Coagulation and Vessel Disease, G780887005
OpenAlex ID: W2088721908
FWCI: 2.472
Cites: 54
Cited by: 63
Related to: 10
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