Work

Deletion of Smad4 in fibroblasts leads to defective chondrocyte maturation and cartilage production in a TGFβ type II receptor independent manner

Year: 2011

Type: article

Source: Biochemical and Biophysical Research Communications

DOI: 10.1016/j.bbrc.2011.02.142

Authors: Yingqi Teng, Keizo Kanasaki, Nabeel Bardeesy, Hikaru Sugimoto, Raghu Kalluri

Institutions: Beth Israel Deaconess Medical Center, Harvard University, Massachusetts General Hospital, Harvard–MIT Division of Health Sciences and Technology

Language: English

Topic: TGF-β signaling in diseases

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: green

Funders: Beth Israel Deaconess Medical Center, National Institutes of Health, National Institute on Alcohol Abuse and Alcoholism, National Institute of Diabetes and Digestive and Kidney Diseases

Awards: G2512559947, G4382730148, Regulation of Angiogenesis by Tumstatin, G5481787692, Matrix Microenvironment and Liver Fibrosis, G632314946, The Biology and Function of BMP-7 in the Kidney, Assembly of Type IV Collagen in Health and Disease

OpenAlex ID: W2090430768

FWCI: 0.389

Cites: 16

Cited by: 13

Related to: 10