In progressive nephropathies, overload of tubular cells with filtered proteins translates glomerular permeability dysfunction into cellular signals of interstitial inflammation.
Year: 1998
Type: article
DOI: 10.1681/asn.v971213
Authors: M Abbate, C Zoja, D Corna, M Capitanio, T Bertani, G Remuzzi
Institution: Mario Negri Institute for Pharmacological Research
Language: English
Subfield: Nephrology
Field: Medicine
Domain: Health Sciences
Open Access status: closed
OpenAlex ID: W2108910108
