Work

Presenilin-1 uses phospholipase D1 as a negative regulator of β-amyloid formation

Year: 2006

Type: article

Source: Proceedings of the National Academy of Sciences

DOI: 10.1073/pnas.0510708103

Authors: Dongming Cai, William J. Netzer, Minghao Zhong, Yixin Lin, Guangwei Du, Michael Frohman, David A. Foster, Sangram S. Sisodia, Huaxi Xu, Fred S. Gorelick, Paul Greengard

Institutions: Rockefeller University, City University of New York, Hunter College, Stony Brook University Hospital, Stony Brook University, University of Chicago, Sanford Burnham Prebys Medical Discovery Institute, Yale University, VA Connecticut Healthcare System

Language: English

Topic: Alzheimer's disease research and treatments

Subfield: Physiology

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: closed

Funders: U.S. Department of Veterans Affairs, Alzheimer's Association, F. M. Kirby Foundation, Fisher Center for Alzheimer's Research Foundation, James N. Kirby Foundation, National Institutes of Health, National Institute on Aging, National Cancer Institute, National Institute of Neurological Disorders and Stroke

Awards: G1156585274, G1431933605, G1837380957, Regulation of BetaAPP Trafficking and Neuronal Functions, Regulation of Protein Trafficking in Neurodegeneration, G3629265633, G447323032, Phospholipase D-mTOR Survival Signals in Tumorigenesis, Signaling Transduction and Alzheimer's Disease, G8365684821

OpenAlex ID: W2117067724

FWCI: 6.866

Cites: 36

Cited by: 111

Related to: 10