Work

Loss of Activin Receptor Type 1B Accelerates Development of Intraductal Papillary Mucinous Neoplasms in Mice With Activated KRAS

Year: 2015

Type: article

Source: Gastroenterology

DOI: 10.1053/j.gastro.2015.09.013

Authors: Wanglong Qiu, Sophia M. Tang, Sohyae Lee, Andrew T. Turk, Anthony N. Sireci, Anne Qiu, Christian Rose, Chuangao Xie, Jan Kitajewski, Hui-Ju Wen, Howard C. Crawford, Peter A. Sims, Ralph H. Hruban, Helen E. Remotti, Gloria H. Su

Institutions: Columbia University Irving Medical Center, Herbert Irving Comprehensive Cancer Center, Mayo Clinic, Mayo Clinic in Florida, Mayo Clinic Comprehensive Cancer Center (Florida), Johns Hopkins University, Johns Hopkins Medicine

Language: English

Topic: Pancreatic and Hepatic Oncology Research

Subfield: Oncology

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: green

Funders: Pancreatic Cancer Action Network, National Cancer Institute

Awards: The tumor-suppressive role of ALK4/ACVR1B in pancreatic tumorigenesis, G3431373237, G3766856912, Mouse Model for Human Pancreatic Ductal Adenocarcinoma, ADAM17 in pancreatic cancer and pancreatitis, DPC4 function in human pancreatic cancer, Mouse Model for Human Pancreatic Ductal Adenocarcinoma, G7003717824, G8672325208

OpenAlex ID: W2119960643

FWCI: 1.606

Cites: 39

Cited by: 39

Related to: 10