Work

FAK Deficiency in Cells Contributing to the Basal Lamina Results in Cortical Abnormalities Resembling Congenital Muscular Dystrophies

Year: 2003

Type: article

Source: Neuron

DOI: 10.1016/s0896-6273(03)00666-4

Authors: Hilary E. Beggs, Dorreyah Schahin-Reed, Keling Zang, Sandra Goebbels, Klaus-Armin Nave, Jessica Gorski, Kevin R. Jones, David Sretavan, Louis F. Reichardt

Institutions: Howard Hughes Medical Institute, University of California, San Francisco, Max Planck Society, Max Planck Institute of Experimental Medicine, University of Colorado Boulder

Language: English

Topic: Neurogenesis and neuroplasticity mechanisms

Subfield: Developmental Neuroscience

Field: Neuroscience

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: bronze

Funders: National Eye Institute, National Institute of Neurological Disorders and Stroke

Awards: Integrin Signaling in Nervous System Development, CONSEQUENCES OF FAK DISRUPTION ON NEURONAL DEVELOPMENT, Enhanced Axon Sparing In Glaucoma Through Augmentation Of EphB/ephrin B Signaling, G8240147926

OpenAlex ID: W2121645463

FWCI: 3.531

Cites: 71

Cited by: 305

Related to: 8