Work

AMPAR Removal Underlies Aβ-Induced Synaptic Depression and Dendritic Spine Loss

Year: 2006

Type: article

Source: Neuron

DOI: 10.1016/j.neuron.2006.10.035

Authors: Helen Hsieh, Jannic Boehm, Chihiro Sato, Takeshi Iwatsubo, Taisuke Tomita, Sangram Sisodia, Roberto Malinow

Institutions: State University of New York, Cold Spring Harbor Laboratory, Stony Brook University, The University of Tokyo, University of Chicago

Language: English

Topic: Neuroscience and Neuropharmacology Research

Subfield: Cellular and Molecular Neuroscience

Field: Neuroscience

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: bronze

Funders: Alzheimer's Association, Fidelity Foundation, Israel National Road Safety Authority, Ministry of Education, Culture, Sports, Science and Technology, National Institute of Biomedical Innovation, National Institutes of Health, National Institute on Aging, National Institute of Mental Health, National Institute of General Medical Sciences, National Institute of Neurological Disorders and Stroke

Awards: Protein Kinases in Synaptic Plasticity, Mechanisms of LTP:AMPA receptor dynamics, Mechanisms of synaptic LTP, Medical Scientist Training Program, Physiology & Pathophysiology of PS1 & FAD-linked Mutants, Novel roles by glutamatergic receptors in the synaptic effects of beta amyloid, G825950733

OpenAlex ID: W2128401893

FWCI: 17.68

Cites: 102

Cited by: 1,042

Related to: 10