Work

Dissociation of raptor from mTOR is a mechanism of rapamycin‐induced inhibition of mTOR function

Year: 2004

Type: article

Source: Genes to Cells

DOI: 10.1111/j.1356-9597.2004.00727.x

Authors: Noriko Oshiro, Ken‐ichi Yoshino, Sujuti Hidayat, Chiharu Tokunaga, Kenta Hara, Satoshi Eguchi, Joseph Avruch, Kazuyoshi Yonezawa

Institutions: Japan Science and Technology Agency, Biosignal Research Center, Kobe University, Kobe University, Harvard University, Massachusetts General Hospital

Language: English

Topic: PI3K/AKT/mTOR signaling in cancer

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: closed

Funders: National Institutes of Health, Japan Society for the Promotion of Science, National Cancer Institute, National Institute of Diabetes and Digestive and Kidney Diseases

Awards: G4462181674, Molecular basis of Cell Growth Control by the TOR kinase

OpenAlex ID: W2129307474

FWCI: 5.1

Cites: 34

Cited by: 317

Related to: 10