Work

PTEN is inversely correlated with the cell survival factor Akt/PKB and is inactivated via multiple mechanismsin haematological malignancies

Year: 1999

Type: article

Source: Human Molecular Genetics

DOI: 10.1093/hmg/8.2.185

Authors: P. Dahia, R C Aguiar, J Alberta, J B Kum, S Caron, H Sill, D J Marsh, J Ritz, A Freedman, C Stiles, C Eng

Institutions: Harvard University, The Ohio State University Comprehensive Cancer Center – Arthur G. James Cancer Hospital and Richard J. Solove Research Institute, Dana-Farber Cancer Institute, The Ohio State University

Language: English

Topic: PI3K/AKT/mTOR signaling in cancer

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: closed

Funders: Susan G. Komen for the Cure, Susan G. Komen

OpenAlex ID: W2130867126

FWCI: 7.792

Cites: 53

Cited by: 279

Related to: 10