Work

Activation of MAPK by TRH Requires Clathrin-Dependent Endocytosis and PKC but Not Receptor Interaction with β-Arrestin or Receptor Endocytosis

Year: 2001

Type: article

Source: Molecular Endocrinology

DOI: 10.1210/mend.15.9.0695

Authors: Jeffrey Smith, Run Yu, Patricia M. Hinkle

Institutions: University of Rochester Medicine, University of Rochester

Language: English

Topic: Receptor Mechanisms and Signaling

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

Open Access status: closed

Funders: University of Virginia, National Cancer Institute, National Institute of Diabetes and Digestive and Kidney Diseases

Awards: G233393104, G2974861299, Modulation of Receptor Number in Cultured Cells

OpenAlex ID: W2132742882

FWCI: 1.303

Cites: 44

Cited by: 32

Related to: 10