Work

Overexpression of a kinase‐inactive ATR protein causes sensitivity to DNA‐damaging agents and defects in cell cycle checkpoints

Year: 1998

Type: article

Source: The EMBO Journal

DOI: 10.1093/emboj/17.1.159

Authors: William A. Cliby, Christopher J. Roberts, Karlene A. Cimprich, Cheri M. Stringer, John R. Lamb, Stuart L. Schreiber, Stephen H. Friend

Institutions: Mayo Clinic, Fred Hutch Cancer Center, Howard Hughes Medical Institute, Harvard University, Stanford Medicine, Stanford University

Language: English

Topic: DNA Repair Mechanisms

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: bronze

Funders: Damon Runyon Cancer Research Foundation, Mayo Foundation for Medical Education and Research

OpenAlex ID: W2133318290

FWCI: 11.14

Cites: 55

Cited by: 543

Related to: 10