Work

Ablation of AMP-activated protein kinase α1 and α2 from mouse pancreatic beta cells and RIP2.Cre neurons suppresses insulin release in vivo

Year: 2010

Type: article

Source: Diabetologia

DOI: 10.1007/s00125-010-1692-1

Authors: G. Sun, A. I. Tarasov, J. McGinty, A. McDonald, G. da Silva Xavier, T. Gorman, A. Marley, P. M. French, H. Parker, F. Gribble, F. Reimann, O. Prendiville, R. Carzaniga, B. Viollet, I. Leclerc, G. A. Rutter

Institutions: Imperial College London, AstraZeneca (United Kingdom), University of Cambridge, Charing Cross Hospital, Centre National de la Recherche Scientifique, Inserm, Institut Cochin, Université Paris Descartes

Language: English

Topic: Pancreatic function and diabetes

Subfield: Surgery

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: bronze

Funders: Queen Mary University of London, Wellcome Trust, Diabetes UK, European Commission, Université de Genève, Université de Lausanne, National Institutes of Health, Medical Research Council, National Institute of Diabetes and Digestive and Kidney Diseases

Awards: G476811148, G4873118862, G5337456671, Molecular mechanisms of glucose sending by pancreatic islet b-cells and hypothalamic neurons., The role of PAS domain containing protein kinase in pancreatic ?-cell function, The role of PAS kinase in hepatic lipid metabolism, Enteroendocrine cell function in diabetes and obesity., Mechanisms underlying Glucose-dependent Insulinotropic Polypeptide (GIP) secretion and function.

OpenAlex ID: W2135362510

FWCI: 12.22

Cites: 56

Cited by: 97

Related to: 10