Year: 2008
Type: article
Abstract: In this issue of Blood, Tiedt and colleagues use transgenic and retroviral models to demonstrate that the expression level of JAK2V617F plays an important role in determining myeloproliferative disease (MPD) phenotype.
Source: Blood
Author: Ross L. Levine
Institution: Memorial Sloan Kettering Cancer Center
Language: English
Subfield: Genetics
Field: Medicine
Domain: Health Sciences
Open Access status: bronze
OpenAlex ID: W2138060606