Work

Epileptogenesis causes an N-methyl-d-aspartate receptor/Ca2+-dependent decrease in Ca2+/calmodulin-dependent protein kinase II activity in a hippocampal neuronal culture model of spontaneous recurrent epileptiform discharges

Year: 2008

Type: article

Source: European Journal of Pharmacology

DOI: 10.1016/j.ejphar.2008.04.021

Authors: Robert E. Blair, Sompong Sombati, Severn B. Churn, Robert J. DeLorenzo

Institution: Virginia Commonwealth University

Language: English

Topic: Epilepsy research and treatment

Subfield: Psychiatry and Mental health

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: green

Funders: National Institutes of Health, National Institute on Drug Abuse, National Institute of Neurological Disorders and Stroke

Awards: G292588079, STATUS EPILEPTICUS REORGANIZES CANNABINOID RECEPTORS, G3856495391, PARATHION EXPOSURE: MECHANISMS OF TOXICTY AND TREATMENT, PATHOPHYSIOLOGY AND MORTALITY OF STATUS EPILEPTICUS, CENTER FOR DRUG ABUSE RESEARCH

OpenAlex ID: W2143664312

FWCI: 1.402

Cites: 49

Cited by: 32

Related to: 10