Work

Mice deficient for the gap junction protein Connexin32 exhibit increased radiation-induced tumorigenesis associated with elevated mitogen-activated protein kinase (p44/Erk1, p42/Erk2) activation

Year: 2004

Type: article

Source: Carcinogenesis

DOI: 10.1093/carcin/bgh071

Authors: Timothy J. King, Paul D. Lampe

Institutions: Fred Hutch Cancer Center, University of Washington

Language: English

Topic: Connexins and lens biology

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: closed

Funders: Fred Hutchinson Cancer Research Center, National Institutes of Health, National Institute of General Medical Sciences, National Institute of Allergy and Infectious Diseases, National Institute of Arthritis and Musculoskeletal and Skin Diseases

Awards: Communication Between Laminin 5 and Gap Junctions, Diseases of Public Health Importance Training Grant, G5019326122, G542351383, Phosphorylation of Gap Junction Proteins

OpenAlex ID: W2145626647

FWCI: 3.62

Cites: 69

Cited by: 100

Related to: 10