Work

Deprivation of MKK7 in cardiomyocytes provokes heart failure in mice when exposed to pressure overload

Year: 2011

Type: article

Source: Journal of Molecular and Cellular Cardiology

DOI: 10.1016/j.yjmcc.2011.01.013

Authors: Wei Liu, Min Zi, Hongbo Chi, Jiawei Jin, Sukhpal Prehar, Ludwig Neyses, Elizabeth J. Cartwright, Richard A. Flavell, Roger J. Davis, Xin Wang

Institutions: University of Manchester, St. Jude Children's Research Hospital, Howard Hughes Medical Institute, Yale University, University of Massachusetts Chan Medical School

Language: English

Topic: Cardiac Fibrosis and Remodeling

Subfield: Cardiology and Cardiovascular Medicine

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: closed

Funders: Foundation for the National Institutes of Health, Howard Hughes Medical Institute, British Heart Foundation, National Cancer Institute

Awards: G1334054042, The signalling regulation of ventricular arrhythmias and cardiac gap junctions in mice with a cardiac-specific deletion of MKK4, The signalling regulation of cardiac hypertrophy in mice with a cardiomyocyte-specifi c deletion of ERK5, Mechanisms of Action of Pro-inflammatory Cytokines, Investigating a novel mechanism of cardiac glucose metabolism regulation in diabetes

OpenAlex ID: W2145941238

FWCI: 1.228

Cites: 42

Cited by: 33

Related to: 10